Therefore, we hypothesized that myosin IIa is normally a poor regulator of BCR signaling. suggest that myosin IIa is normally a poor regulator of B cell activation but an optimistic
ROCK inhibitors
Therefore, we hypothesized that myosin IIa is normally a poor regulator of BCR signaling. suggest that myosin IIa is normally a poor regulator of B cell activation but an optimistic