Hoch et al. antibody-mediated autoimmune disease in which the nicotinic acetylcholine receptor (AChR) at neuromuscular junctions (NMJs) is the major autoantigen (1). AChR-specific antibodies are detected in 90% of nonimmunosuppressed

Scale bars, 50 m. signaling pathways in cells with TMEM176B silencing and overexpression. Silencing TMEM176B or inhibiting it having a restorative antibody impaired cell proliferation, while overexpression improved proliferation in